Electrophilic PPARγ Ligands Attenuate IL-1β and Silica-Induced Inflammatory Mediator Production in Human Lung Fibroblasts via a PPARγ-Independent Mechanism.

Hogan CM, Thatcher TH, Sapinoro RE, Gurell MN, Ferguson HE, Pollock SJ, Jones C, Phipps RP, Sime PJ

Open source

DOI
10.1155/2011/318134
Published
2011
Container
PPAR research
Publisher
Not recorded
Open access
yes

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BibTeX

@article{allodium:10.1155/2011/318134,
  title = {Electrophilic PPARγ Ligands Attenuate IL-1β and Silica-Induced Inflammatory Mediator Production in Human Lung Fibroblasts via a PPARγ-Independent Mechanism.},
  author = {Hogan CM and Thatcher TH and Sapinoro RE and Gurell MN and Ferguson HE and Pollock SJ and Jones C and Phipps RP and Sime PJ},
  year = {2011},
  journal = {PPAR research},
  doi = {10.1155/2011/318134},
  url = {https://doi.org/10.1155/2011/318134}
}

RIS

TY  - JOUR
TI  - Electrophilic PPARγ Ligands Attenuate IL-1β and Silica-Induced Inflammatory Mediator Production in Human Lung Fibroblasts via a PPARγ-Independent Mechanism.
AU  - Hogan CM
AU  - Thatcher TH
AU  - Sapinoro RE
AU  - Gurell MN
AU  - Ferguson HE
AU  - Pollock SJ
AU  - Jones C
AU  - Phipps RP
AU  - Sime PJ
PY  - 2011
JO  - PPAR research
DO  - 10.1155/2011/318134
UR  - https://doi.org/10.1155/2011/318134
ER  - 

APA

CM, H., TH, T., RE, S., MN, G., HE, F., SJ, P., C, J., RP, P., & PJ, S. (2011). Electrophilic PPARγ Ligands Attenuate IL-1β and Silica-Induced Inflammatory Mediator Production in Human Lung Fibroblasts via a PPARγ-Independent Mechanism.. PPAR research. https://doi.org/10.1155/2011/318134

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