The Natural Compound Dehydrocrenatidine Attenuates Nicotine-Induced Stemness and Epithelial-Mesenchymal Transition in Hepatocellular Carcinoma by Regulating a7nAChR-Jak2 Signaling Pathways.
- DOI
- 10.1155/2022/8316335
- Published
- 2022
- Container
- Disease markers
- Publisher
- Not recorded
- Open access
- yes
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Cite this work
BibTeX
@article{allodium:10.1155/2022/8316335,
title = {The Natural Compound Dehydrocrenatidine Attenuates Nicotine-Induced Stemness and Epithelial-Mesenchymal Transition in Hepatocellular Carcinoma by Regulating a7nAChR-Jak2 Signaling Pathways.},
author = {Li CL and Wang CC and Pai HT and Tu SL and Hou PY and Huang CY and Huang MT and Chang YJ},
year = {2022},
journal = {Disease markers},
doi = {10.1155/2022/8316335},
url = {https://doi.org/10.1155/2022/8316335}
}RIS
TY - JOUR TI - The Natural Compound Dehydrocrenatidine Attenuates Nicotine-Induced Stemness and Epithelial-Mesenchymal Transition in Hepatocellular Carcinoma by Regulating a7nAChR-Jak2 Signaling Pathways. AU - Li CL AU - Wang CC AU - Pai HT AU - Tu SL AU - Hou PY AU - Huang CY AU - Huang MT AU - Chang YJ PY - 2022 JO - Disease markers DO - 10.1155/2022/8316335 UR - https://doi.org/10.1155/2022/8316335 ER -
APA
CL, L., CC, W., HT, P., SL, T., PY, H., CY, H., MT, H., & YJ, C. (2022). The Natural Compound Dehydrocrenatidine Attenuates Nicotine-Induced Stemness and Epithelial-Mesenchymal Transition in Hepatocellular Carcinoma by Regulating a7nAChR-Jak2 Signaling Pathways.. Disease markers. https://doi.org/10.1155/2022/8316335
Source records
- pubmed · retrieved 2026-09-25T13:49:59.626Z