Induction of cell cycle arrest and apoptosis by the proteasome inhibitor PS-341 in Hodgkin disease cell lines is independent of inhibitor of nuclear factor-kappaB mutations or activation of the CD30, CD40, and RANK receptors.

Zheng B, Georgakis GV, Li Y, Bharti A, McConkey D, Aggarwal BB, Younes A

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DOI
10.1158/1078-0432.ccr-03-0494
Published
2004 May 1
Container
Clinical cancer research : an official journal of the American Association for Cancer Research
Publisher
Not recorded
Open access
unknown

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BibTeX

@article{allodium:10.1158/1078-0432.ccr-03-0494,
  title = {Induction of cell cycle arrest and apoptosis by the proteasome inhibitor PS-341 in Hodgkin disease cell lines is independent of inhibitor of nuclear factor-kappaB mutations or activation of the CD30, CD40, and RANK receptors.},
  author = {Zheng B and Georgakis GV and Li Y and Bharti A and McConkey D and Aggarwal BB and Younes A},
  year = {2004},
  journal = {Clinical cancer research : an official journal of the American Association for Cancer Research},
  doi = {10.1158/1078-0432.ccr-03-0494},
  url = {https://doi.org/10.1158/1078-0432.ccr-03-0494}
}

RIS

TY  - JOUR
TI  - Induction of cell cycle arrest and apoptosis by the proteasome inhibitor PS-341 in Hodgkin disease cell lines is independent of inhibitor of nuclear factor-kappaB mutations or activation of the CD30, CD40, and RANK receptors.
AU  - Zheng B
AU  - Georgakis GV
AU  - Li Y
AU  - Bharti A
AU  - McConkey D
AU  - Aggarwal BB
AU  - Younes A
PY  - 2004
JO  - Clinical cancer research : an official journal of the American Association for Cancer Research
DO  - 10.1158/1078-0432.ccr-03-0494
UR  - https://doi.org/10.1158/1078-0432.ccr-03-0494
ER  - 

APA

B, Z., GV, G., Y, L., A, B., D, M., BB, A., & A, Y. (2004). Induction of cell cycle arrest and apoptosis by the proteasome inhibitor PS-341 in Hodgkin disease cell lines is independent of inhibitor of nuclear factor-kappaB mutations or activation of the CD30, CD40, and RANK receptors.. Clinical cancer research : an official journal of the American Association for Cancer Research. https://doi.org/10.1158/1078-0432.ccr-03-0494

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