CRISPR/Cas9 loss-of-function screen in a neuronal model of AP-4 deficiency identifies ATG9A trafficking modulators.
- DOI
- 10.1172/jci.insight.202204
- Published
- 2026 Jul 22
- Container
- JCI insight
- Publisher
- Not recorded
- Open access
- yes
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Cite this work
BibTeX
@article{allodium:10.1172/jci.insight.202204,
title = {CRISPR/Cas9 loss-of-function screen in a neuronal model of AP-4 deficiency identifies ATG9A trafficking modulators.},
author = {Ziegler M and Günter C and Alecu JE and Xue X and Kim HM and Saffari A and Davies AK and Sahin M and Ebrahimi-Fakhari D},
year = {2026},
journal = {JCI insight},
doi = {10.1172/jci.insight.202204},
url = {https://doi.org/10.1172/jci.insight.202204}
}RIS
TY - JOUR TI - CRISPR/Cas9 loss-of-function screen in a neuronal model of AP-4 deficiency identifies ATG9A trafficking modulators. AU - Ziegler M AU - Günter C AU - Alecu JE AU - Xue X AU - Kim HM AU - Saffari A AU - Davies AK AU - Sahin M AU - Ebrahimi-Fakhari D PY - 2026 JO - JCI insight DO - 10.1172/jci.insight.202204 UR - https://doi.org/10.1172/jci.insight.202204 ER -
APA
M, Z., C, G., JE, A., X, X., HM, K., A, S., AK, D., M, S., & D, E. (2026). CRISPR/Cas9 loss-of-function screen in a neuronal model of AP-4 deficiency identifies ATG9A trafficking modulators.. JCI insight. https://doi.org/10.1172/jci.insight.202204
Source records
- pubmed · retrieved 2026-09-26T17:35:05.006Z