CRISPR/Cas9-induced double-strand breaks in the huntingtin locus lead to CAG repeat contraction through DNA end resection and homology-mediated repair.
- DOI
- 10.1186/s12915-024-02079-6
- Published
- 2024 Dec 3
- Container
- BMC biology
- Publisher
- Not recorded
- Open access
- yes
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Cite this work
BibTeX
@article{allodium:10.1186/s12915-024-02079-6,
title = {CRISPR/Cas9-induced double-strand breaks in the huntingtin locus lead to CAG repeat contraction through DNA end resection and homology-mediated repair.},
author = {Sledzinski P and Nowaczyk M and Smielowska MI and Olejniczak M},
year = {2024},
journal = {BMC biology},
doi = {10.1186/s12915-024-02079-6},
url = {https://doi.org/10.1186/s12915-024-02079-6}
}RIS
TY - JOUR TI - CRISPR/Cas9-induced double-strand breaks in the huntingtin locus lead to CAG repeat contraction through DNA end resection and homology-mediated repair. AU - Sledzinski P AU - Nowaczyk M AU - Smielowska MI AU - Olejniczak M PY - 2024 JO - BMC biology DO - 10.1186/s12915-024-02079-6 UR - https://doi.org/10.1186/s12915-024-02079-6 ER -
APA
P, S., M, N., MI, S., & M, O. (2024). CRISPR/Cas9-induced double-strand breaks in the huntingtin locus lead to CAG repeat contraction through DNA end resection and homology-mediated repair.. BMC biology. https://doi.org/10.1186/s12915-024-02079-6
Source records
- pubmed · retrieved 2026-09-25T10:31:25.736Z