CRISPR/Cas9-induced double-strand breaks in the huntingtin locus lead to CAG repeat contraction through DNA end resection and homology-mediated repair
- DOI
- 10.1186/s12915-024-02079-6
- Published
- 2024-12-03
- Container
- BMC Biology
- Publisher
- Springer Science and Business Media LLC
- Open access
- unknown
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Cite this work
BibTeX
@article{allodium:10.1186/s12915-024-02079-6,
title = {CRISPR/Cas9-induced double-strand breaks in the huntingtin locus lead to CAG repeat contraction through DNA end resection and homology-mediated repair},
author = {Pawel Sledzinski and Mateusz Nowaczyk and Marianna Iga Smielowska and Marta Olejniczak},
year = {2024},
journal = {BMC Biology},
doi = {10.1186/s12915-024-02079-6},
url = {https://doi.org/10.1186/s12915-024-02079-6}
}RIS
TY - JOUR TI - CRISPR/Cas9-induced double-strand breaks in the huntingtin locus lead to CAG repeat contraction through DNA end resection and homology-mediated repair AU - Pawel Sledzinski AU - Mateusz Nowaczyk AU - Marianna Iga Smielowska AU - Marta Olejniczak PY - 2024 JO - BMC Biology DO - 10.1186/s12915-024-02079-6 UR - https://doi.org/10.1186/s12915-024-02079-6 ER -
APA
Sledzinski, P., Nowaczyk, M., Smielowska, M. I., & Olejniczak, M. (2024). CRISPR/Cas9-induced double-strand breaks in the huntingtin locus lead to CAG repeat contraction through DNA end resection and homology-mediated repair. BMC Biology. https://doi.org/10.1186/s12915-024-02079-6
Source records
- crossref · retrieved 2026-11-09T19:17:52.681Z