Unlocking thalidomide response as a hemoglobin F augmentation agent in transfusion-dependent β-thalassemia: the genetic impact of BCL11A, HBS1L-MYB, and XmnI polymorphism
- DOI
- 10.1186/s13000-026-01797-5
- Published
- 2026-07-07
- Container
- Diagnostic Pathology
- Publisher
- Springer Science and Business Media LLC
- Open access
- unknown
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Cite this work
BibTeX
@article{allodium:10.1186/s13000-026-01797-5,
title = {Unlocking thalidomide response as a hemoglobin F augmentation agent in transfusion-dependent β-thalassemia: the genetic impact of BCL11A, HBS1L-MYB, and XmnI polymorphism},
author = {Iram Nazir and Muhammad Younus Jamal Siddiqi and Maeesa Wadood and Muhammad Rizwan},
year = {2026},
journal = {Diagnostic Pathology},
doi = {10.1186/s13000-026-01797-5},
url = {https://doi.org/10.1186/s13000-026-01797-5}
}RIS
TY - JOUR TI - Unlocking thalidomide response as a hemoglobin F augmentation agent in transfusion-dependent β-thalassemia: the genetic impact of BCL11A, HBS1L-MYB, and XmnI polymorphism AU - Iram Nazir AU - Muhammad Younus Jamal Siddiqi AU - Maeesa Wadood AU - Muhammad Rizwan PY - 2026 JO - Diagnostic Pathology DO - 10.1186/s13000-026-01797-5 UR - https://doi.org/10.1186/s13000-026-01797-5 ER -
APA
Nazir, I., Siddiqi, M. Y. J., Wadood, M., & Rizwan, M. (2026). Unlocking thalidomide response as a hemoglobin F augmentation agent in transfusion-dependent β-thalassemia: the genetic impact of BCL11A, HBS1L-MYB, and XmnI polymorphism. Diagnostic Pathology. https://doi.org/10.1186/s13000-026-01797-5
Source records
- crossref · retrieved 2026-09-26T07:03:58.417Z