Complement 3(+)-astrocytes are highly abundant in prion diseases, but their abolishment led to an accelerated disease course and early dysregulation of microglia.
- DOI
- 10.1186/s40478-019-0735-1
- Published
- 2019 May 22
- Container
- Acta neuropathologica communications
- Publisher
- Not recorded
- Open access
- yes
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Cite this work
BibTeX
@article{allodium:10.1186/s40478-019-0735-1,
title = {Complement 3(+)-astrocytes are highly abundant in prion diseases, but their abolishment led to an accelerated disease course and early dysregulation of microglia.},
author = {Hartmann K and Sepulveda-Falla D and Rose IVL and Madore C and Muth C and Matschke J and Butovsky O and Liddelow S and Glatzel M and Krasemann S},
year = {2019},
journal = {Acta neuropathologica communications},
doi = {10.1186/s40478-019-0735-1},
url = {https://doi.org/10.1186/s40478-019-0735-1}
}RIS
TY - JOUR TI - Complement 3(+)-astrocytes are highly abundant in prion diseases, but their abolishment led to an accelerated disease course and early dysregulation of microglia. AU - Hartmann K AU - Sepulveda-Falla D AU - Rose IVL AU - Madore C AU - Muth C AU - Matschke J AU - Butovsky O AU - Liddelow S AU - Glatzel M AU - Krasemann S PY - 2019 JO - Acta neuropathologica communications DO - 10.1186/s40478-019-0735-1 UR - https://doi.org/10.1186/s40478-019-0735-1 ER -
APA
K, H., D, S., IVL, R., C, M., C, M., J, M., O, B., S, L., M, G., & S, K. (2019). Complement 3(+)-astrocytes are highly abundant in prion diseases, but their abolishment led to an accelerated disease course and early dysregulation of microglia.. Acta neuropathologica communications. https://doi.org/10.1186/s40478-019-0735-1
Source records
- pubmed · retrieved 2026-09-26T11:11:32.286Z
- europe-pmc · retrieved 2026-09-26T11:11:32.297Z
- doaj · retrieved 2026-09-26T11:11:32.280Z
- hal · retrieved 2026-09-26T11:11:32.321Z