High reelin expression may explain why a subgroup of entorhinal cortex neurons functions as an initial nucleation site of Alzheimer's disease.

Kobro-Flatmoen A, Hussan JR, Hunter PJ, Omholt SW

DOI: 10.1371/journal.pcbi.1014532
Published: 2026 Aug
Container: PLoS computational biology
Publisher: Not recorded
Open access: yes

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Cite this work

BibTeX

@article{allodium:10.1371/journal.pcbi.1014532,
  title = {High reelin expression may explain why a subgroup of entorhinal cortex neurons functions as an initial nucleation site of Alzheimer's disease.},
  author = {Kobro-Flatmoen A and Hussan JR and Hunter PJ and Omholt SW},
  year = {2026},
  journal = {PLoS computational biology},
  doi = {10.1371/journal.pcbi.1014532},
  url = {https://doi.org/10.1371/journal.pcbi.1014532}
}

RIS

TY  - JOUR
TI  - High reelin expression may explain why a subgroup of entorhinal cortex neurons functions as an initial nucleation site of Alzheimer's disease.
AU  - Kobro-Flatmoen A
AU  - Hussan JR
AU  - Hunter PJ
AU  - Omholt SW
PY  - 2026
JO  - PLoS computational biology
DO  - 10.1371/journal.pcbi.1014532
UR  - https://doi.org/10.1371/journal.pcbi.1014532
ER  -

APA

A, K., JR, H., PJ, H., & SW, O. (2026). High reelin expression may explain why a subgroup of entorhinal cortex neurons functions as an initial nucleation site of Alzheimer's disease.. PLoS computational biology. https://doi.org/10.1371/journal.pcbi.1014532

Source records

pubmed · retrieved 2026-09-26T18:18:57.815Z