Neuroinflammation and ER-stress are key mechanisms of acute bilirubin toxicity and hearing loss in a mouse model
- DOI
- 10.1371/journal.pone.0201022
- Published
- 2018-08-14
- Container
- PLOS ONE
- Publisher
- Public Library of Science (PLoS)
- Open access
- unknown
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Cite this work
BibTeX
@article{allodium:10.1371/journal.pone.0201022,
title = {Neuroinflammation and ER-stress are key mechanisms of acute bilirubin toxicity and hearing loss in a mouse model},
author = {Emanuele Schiavon and Joshua L. Smalley and Sherylanne Newton and Nigel H. Greig and Ian D. Forsythe},
year = {2018},
journal = {PLOS ONE},
doi = {10.1371/journal.pone.0201022},
url = {https://doi.org/10.1371/journal.pone.0201022}
}RIS
TY - JOUR TI - Neuroinflammation and ER-stress are key mechanisms of acute bilirubin toxicity and hearing loss in a mouse model AU - Emanuele Schiavon AU - Joshua L. Smalley AU - Sherylanne Newton AU - Nigel H. Greig AU - Ian D. Forsythe PY - 2018 JO - PLOS ONE DO - 10.1371/journal.pone.0201022 UR - https://doi.org/10.1371/journal.pone.0201022 ER -
APA
Schiavon, E., Smalley, J. L., Newton, S., Greig, N. H., & Forsythe, I. D. (2018). Neuroinflammation and ER-stress are key mechanisms of acute bilirubin toxicity and hearing loss in a mouse model. PLOS ONE. https://doi.org/10.1371/journal.pone.0201022
Source records
- crossref · retrieved 2026-09-26T00:30:54.226Z