Familial Alzheimer’s disease mutations at position 22 of the amyloid β-peptide sequence differentially affect synaptic loss, tau phosphorylation and neuronal cell death in an ex vivo system

Christian Tackenberg, Luka Kulic, Roger M. Nitsch

Open source

DOI
10.1371/journal.pone.0239584
Published
2020-09-23
Container
PLOS ONE
Publisher
Public Library of Science (PLoS)
Open access
unknown

Credibility signals

uncertain Score 64/100 under policy 1.0.0. This is a metadata assessment, not a judgment of the paper's conclusions.

Show all credibility signals

Cite this work

BibTeX

@article{allodium:10.1371/journal.pone.0239584,
  title = {Familial Alzheimer’s disease mutations at position 22 of the amyloid β-peptide sequence differentially affect synaptic loss, tau phosphorylation and neuronal cell death in an ex vivo system},
  author = {Christian Tackenberg and Luka Kulic and Roger M. Nitsch},
  year = {2020},
  journal = {PLOS ONE},
  doi = {10.1371/journal.pone.0239584},
  url = {https://doi.org/10.1371/journal.pone.0239584}
}

RIS

TY  - JOUR
TI  - Familial Alzheimer’s disease mutations at position 22 of the amyloid β-peptide sequence differentially affect synaptic loss, tau phosphorylation and neuronal cell death in an ex vivo system
AU  - Christian Tackenberg
AU  - Luka Kulic
AU  - Roger M. Nitsch
PY  - 2020
JO  - PLOS ONE
DO  - 10.1371/journal.pone.0239584
UR  - https://doi.org/10.1371/journal.pone.0239584
ER  - 

APA

Tackenberg, C., Kulic, L., & Nitsch, R. M. (2020). Familial Alzheimer’s disease mutations at position 22 of the amyloid β-peptide sequence differentially affect synaptic loss, tau phosphorylation and neuronal cell death in an ex vivo system. PLOS ONE. https://doi.org/10.1371/journal.pone.0239584

Source records