Molecular docking and mouse modeling suggest CMKLR1 and INSR as targets for improving PCOS phenotypes by minocycline.

Kian M, Hosseini E, Abdizadeh T, Langaee T, Khajouei A, Ghasemi S

DOI: 10.17179/excli2021-4534
Published: 2022
Container: EXCLI journal
Publisher: Not recorded
Open access: yes

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Cite this work

BibTeX

@article{allodium:10.17179/excli2021-4534,
  title = {Molecular docking and mouse modeling suggest CMKLR1 and INSR as targets for improving PCOS phenotypes by minocycline.},
  author = {Kian M and Hosseini E and Abdizadeh T and Langaee T and Khajouei A and Ghasemi S},
  year = {2022},
  journal = {EXCLI journal},
  doi = {10.17179/excli2021-4534},
  url = {https://doi.org/10.17179/excli2021-4534}
}

RIS

TY  - JOUR
TI  - Molecular docking and mouse modeling suggest CMKLR1 and INSR as targets for improving PCOS phenotypes by minocycline.
AU  - Kian M
AU  - Hosseini E
AU  - Abdizadeh T
AU  - Langaee T
AU  - Khajouei A
AU  - Ghasemi S
PY  - 2022
JO  - EXCLI journal
DO  - 10.17179/excli2021-4534
UR  - https://doi.org/10.17179/excli2021-4534
ER  -

APA

M, K., E, H., T, A., T, L., A, K., & S, G. (2022). Molecular docking and mouse modeling suggest CMKLR1 and INSR as targets for improving PCOS phenotypes by minocycline.. EXCLI journal. https://doi.org/10.17179/excli2021-4534

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pubmed · retrieved 2026-09-25T08:57:35.577Z