Activation of endoplasmic reticulum stress promotes autophagy and apoptosis and reverses chemoresistance of human small cell lung cancer cells by inhibiting the PI3K/AKT/mTOR signaling pathway
- DOI
- 10.18632/oncotarget.12718
- Published
- 2016-10-18
- Container
- Oncotarget
- Publisher
- Impact Journals, LLC
- Open access
- unknown
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Cite this work
BibTeX
@article{allodium:10.18632/oncotarget.12718,
title = {Activation of endoplasmic reticulum stress promotes autophagy and apoptosis and reverses chemoresistance of human small cell lung cancer cells by inhibiting the PI3K/AKT/mTOR signaling pathway},
author = {Xin-Shuang Yu and Juan Du and Yu-Jun Fan and Feng-Jun Liu and Li-Li Cao and Ning Liang and De-Guo Xu and Jian-Dong Zhang},
year = {2016},
journal = {Oncotarget},
doi = {10.18632/oncotarget.12718},
url = {https://doi.org/10.18632/oncotarget.12718}
}RIS
TY - JOUR TI - Activation of endoplasmic reticulum stress promotes autophagy and apoptosis and reverses chemoresistance of human small cell lung cancer cells by inhibiting the PI3K/AKT/mTOR signaling pathway AU - Xin-Shuang Yu AU - Juan Du AU - Yu-Jun Fan AU - Feng-Jun Liu AU - Li-Li Cao AU - Ning Liang AU - De-Guo Xu AU - Jian-Dong Zhang PY - 2016 JO - Oncotarget DO - 10.18632/oncotarget.12718 UR - https://doi.org/10.18632/oncotarget.12718 ER -
APA
Yu, X., Du, J., Fan, Y., Liu, F., Cao, L., Liang, N., Xu, D., & Zhang, J. (2016). Activation of endoplasmic reticulum stress promotes autophagy and apoptosis and reverses chemoresistance of human small cell lung cancer cells by inhibiting the PI3K/AKT/mTOR signaling pathway. Oncotarget. https://doi.org/10.18632/oncotarget.12718
Source records
- crossref · retrieved 2026-09-24T23:28:00.172Z