The α7 Nicotinic Acetylcholine Receptor Agonist GTS-21 Improves Bacterial Clearance in Mice by Restoring Hyperoxia-Compromised Macrophage Function
- DOI
- 10.2119/molmed.2013.00086
- Published
- 2014-01
- Container
- Molecular Medicine
- Publisher
- Springer Science and Business Media LLC
- Open access
- unknown
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Cite this work
BibTeX
@article{allodium:10.2119/molmed.2013.00086,
title = {The α7 Nicotinic Acetylcholine Receptor Agonist GTS-21 Improves Bacterial Clearance in Mice by Restoring Hyperoxia-Compromised Macrophage Function},
author = {Ravikumar A. Sitapara and Daniel J. Antoine and Lokesh Sharma and Vivek S. Patel and Charles R. Ashby and Samir Gorasiya and Huan Yang and Michelle Zur and Lin L. Mantell},
year = {2014},
journal = {Molecular Medicine},
doi = {10.2119/molmed.2013.00086},
url = {https://doi.org/10.2119/molmed.2013.00086}
}RIS
TY - JOUR TI - The α7 Nicotinic Acetylcholine Receptor Agonist GTS-21 Improves Bacterial Clearance in Mice by Restoring Hyperoxia-Compromised Macrophage Function AU - Ravikumar A. Sitapara AU - Daniel J. Antoine AU - Lokesh Sharma AU - Vivek S. Patel AU - Charles R. Ashby AU - Samir Gorasiya AU - Huan Yang AU - Michelle Zur AU - Lin L. Mantell PY - 2014 JO - Molecular Medicine DO - 10.2119/molmed.2013.00086 UR - https://doi.org/10.2119/molmed.2013.00086 ER -
APA
Sitapara, R. A., Antoine, D. J., Sharma, L., Patel, V. S., Ashby, C. R., Gorasiya, S., Yang, H., Zur, M., & Mantell, L. L. (2014). The α7 Nicotinic Acetylcholine Receptor Agonist GTS-21 Improves Bacterial Clearance in Mice by Restoring Hyperoxia-Compromised Macrophage Function. Molecular Medicine. https://doi.org/10.2119/molmed.2013.00086
Source records
- crossref · retrieved 2026-09-26T02:24:07.093Z