Overexpression of miR-150 alleviates mechanical stress-accelerated the apoptosis of chondrocytes via targeting GRP94.
- DOI
- 10.26355/eurrev_201909_18987
- Published
- 2019 Sep
- Container
- European review for medical and pharmacological sciences
- Publisher
- Not recorded
- Open access
- unknown
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Cite this work
BibTeX
@article{allodium:10.26355/eurrev_201909_18987,
title = {Overexpression of miR-150 alleviates mechanical stress-accelerated the apoptosis of chondrocytes via targeting GRP94.},
author = {Zhang ZQ and Wang CS and Yang P and Wang KZ},
year = {2019},
journal = {European review for medical and pharmacological sciences},
doi = {10.26355/eurrev_201909_18987},
url = {https://doi.org/10.26355/eurrev_201909_18987}
}RIS
TY - JOUR TI - Overexpression of miR-150 alleviates mechanical stress-accelerated the apoptosis of chondrocytes via targeting GRP94. AU - Zhang ZQ AU - Wang CS AU - Yang P AU - Wang KZ PY - 2019 JO - European review for medical and pharmacological sciences DO - 10.26355/eurrev_201909_18987 UR - https://doi.org/10.26355/eurrev_201909_18987 ER -
APA
ZQ, Z., CS, W., P, Y., & KZ, W. (2019). Overexpression of miR-150 alleviates mechanical stress-accelerated the apoptosis of chondrocytes via targeting GRP94.. European review for medical and pharmacological sciences. https://doi.org/10.26355/eurrev_201909_18987
Source records
- pubmed · retrieved 2026-09-25T21:52:50.844Z