Rivaroxaban suppresses factor Xa-Driven PAR-2-ERK inflammatory, catabolic, osteoclastogenic, and mitochondrial dysfunction signaling in osteoarthritis-relevant chondrocytes: a drug-repurposing strategy for joint inflammation.
- DOI
- 10.3389/fphar.2026.1872924
- Published
- 2026
- Container
- Frontiers in pharmacology
- Publisher
- Not recorded
- Open access
- yes
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Cite this work
BibTeX
@article{allodium:10.3389/fphar.2026.1872924,
title = {Rivaroxaban suppresses factor Xa-Driven PAR-2-ERK inflammatory, catabolic, osteoclastogenic, and mitochondrial dysfunction signaling in osteoarthritis-relevant chondrocytes: a drug-repurposing strategy for joint inflammation.},
author = {Patnaik R and Jannati S and Kellett CF and Hing CB and Banerjee Y},
year = {2026},
journal = {Frontiers in pharmacology},
doi = {10.3389/fphar.2026.1872924},
url = {https://doi.org/10.3389/fphar.2026.1872924}
}RIS
TY - JOUR TI - Rivaroxaban suppresses factor Xa-Driven PAR-2-ERK inflammatory, catabolic, osteoclastogenic, and mitochondrial dysfunction signaling in osteoarthritis-relevant chondrocytes: a drug-repurposing strategy for joint inflammation. AU - Patnaik R AU - Jannati S AU - Kellett CF AU - Hing CB AU - Banerjee Y PY - 2026 JO - Frontiers in pharmacology DO - 10.3389/fphar.2026.1872924 UR - https://doi.org/10.3389/fphar.2026.1872924 ER -
APA
R, P., S, J., CF, K., CB, H., & Y, B. (2026). Rivaroxaban suppresses factor Xa-Driven PAR-2-ERK inflammatory, catabolic, osteoclastogenic, and mitochondrial dysfunction signaling in osteoarthritis-relevant chondrocytes: a drug-repurposing strategy for joint inflammation.. Frontiers in pharmacology. https://doi.org/10.3389/fphar.2026.1872924
Source records
- pubmed · retrieved 2026-09-25T20:51:50.194Z
- europe-pmc · retrieved 2026-09-25T20:51:50.224Z
- doaj · retrieved 2026-09-25T20:51:50.210Z