Mechanisms of Fetal Overgrowth in Gestational Diabetes: The Potential Role of SOCS2.

Hernández-Baraza L, Brito-Casillas Y, Valverde-Tercedor C, Recio C, Fernández-Pérez L, Guerra B, Wägner AM

DOI: 10.3390/nu17091519
Published: 2025 Apr 30
Container: Nutrients
Publisher: Not recorded
Open access: yes

Credibility signals

limited evidence Score 45/100 under policy 1.0.0. This is a metadata assessment, not a judgment of the paper's conclusions.

Show all credibility signals

cautionDOI registered: No matching Crossref record was present in this response. Source: Crossref; license: CC0 metadata
cautionDOI resolves: No matching Crossref record was present in this response. Source: Crossref; license: CC0 metadata
not scoredDirectory of Open Access Journals: No matching DOAJ record was present in this response. No allow-list match; this is not evidence of low credibility. Source: DOAJ; license: CC0
not scoredMEDLINE indexed: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
not scoredOpenAlex core source: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
not scoredKnown publisher allow-list: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
not scoredROR affiliation: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
not scoredRetraction Watch retraction: No retraction notice matched this DOI in the deployed snapshot. No matching event found; coverage may be incomplete. Source: Retraction Watch; license: CC BY 4.0
not scoredRetraction Watch expression of concern: No expression of concern notice matched this DOI in the deployed snapshot. No matching event found; coverage may be incomplete. Source: Retraction Watch; license: CC BY 4.0
not scoredRetraction Watch correction: No correction notice matched this DOI in the deployed snapshot. No matching event found; coverage may be incomplete. Source: Retraction Watch; license: CC BY 4.0
not scoredRetraction Watch reinstatement: No reinstatement notice matched this DOI in the deployed snapshot. No matching event found; coverage may be incomplete. Source: Retraction Watch; license: CC BY 4.0
supportingOpen access status: Normalized open-access status: open. Source: Normalized work metadata; license: Caller-provided; provenance license not supplied to scorer
not scoredPublication license: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
not scoredPublication version: A publication version was supplied but is not scored. Source: Normalized work metadata; license: Caller-provided; provenance license not supplied to scorer
cautionMetadata completeness: 5 of 6 scored descriptive metadata groups are present; missing fields increase uncertainty. Source: Normalized work metadata; license: Caller-provided; provenance license not supplied to scorer

Cite this work

BibTeX

@article{allodium:10.3390/nu17091519,
  title = {Mechanisms of Fetal Overgrowth in Gestational Diabetes: The Potential Role of SOCS2.},
  author = {Hernández-Baraza L and Brito-Casillas Y and Valverde-Tercedor C and Recio C and Fernández-Pérez L and Guerra B and Wägner AM},
  year = {2025},
  journal = {Nutrients},
  doi = {10.3390/nu17091519},
  url = {https://doi.org/10.3390/nu17091519}
}

RIS

TY  - JOUR
TI  - Mechanisms of Fetal Overgrowth in Gestational Diabetes: The Potential Role of SOCS2.
AU  - Hernández-Baraza L
AU  - Brito-Casillas Y
AU  - Valverde-Tercedor C
AU  - Recio C
AU  - Fernández-Pérez L
AU  - Guerra B
AU  - Wägner AM
PY  - 2025
JO  - Nutrients
DO  - 10.3390/nu17091519
UR  - https://doi.org/10.3390/nu17091519
ER  -

APA

L, H., Y, B., C, V., C, R., L, F., B, G., & AM, W. (2025). Mechanisms of Fetal Overgrowth in Gestational Diabetes: The Potential Role of SOCS2.. Nutrients. https://doi.org/10.3390/nu17091519

Source records

pubmed · retrieved 2026-09-26T02:38:27.731Z