Suppression of oxidative stress in endothelial progenitor cells promotes angiogenesis and improves cardiac function following myocardial infarction in diabetic mice.

Jin P, Li T, Li X, Shen X, Zhao Y

Open source

DOI
10.3892/etm.2016.3236
Published
2016 Jun
Container
Experimental and therapeutic medicine
Publisher
Not recorded
Open access
yes

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BibTeX

@article{allodium:10.3892/etm.2016.3236,
  title = {Suppression of oxidative stress in endothelial progenitor cells promotes angiogenesis and improves cardiac function following myocardial infarction in diabetic mice.},
  author = {Jin P and Li T and Li X and Shen X and Zhao Y},
  year = {2016},
  journal = {Experimental and therapeutic medicine},
  doi = {10.3892/etm.2016.3236},
  url = {https://doi.org/10.3892/etm.2016.3236}
}

RIS

TY  - JOUR
TI  - Suppression of oxidative stress in endothelial progenitor cells promotes angiogenesis and improves cardiac function following myocardial infarction in diabetic mice.
AU  - Jin P
AU  - Li T
AU  - Li X
AU  - Shen X
AU  - Zhao Y
PY  - 2016
JO  - Experimental and therapeutic medicine
DO  - 10.3892/etm.2016.3236
UR  - https://doi.org/10.3892/etm.2016.3236
ER  - 

APA

P, J., T, L., X, L., X, S., & Y, Z. (2016). Suppression of oxidative stress in endothelial progenitor cells promotes angiogenesis and improves cardiac function following myocardial infarction in diabetic mice.. Experimental and therapeutic medicine. https://doi.org/10.3892/etm.2016.3236

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