Th17/IL-17 may promote ferroptosis through modulation of the TP53/Nrf2 signaling axis in acute-on-chronic liver failure
- DOI
- 10.5114/ceh.2026.164208
- Published
- 2026
- Container
- Clinical and Experimental Hepatology
- Publisher
- Termedia Sp. z.o.o.
- Open access
- unknown
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Cite this work
BibTeX
@article{allodium:10.5114/ceh.2026.164208,
title = {Th17/IL-17 may promote ferroptosis through modulation of the TP53/Nrf2 signaling axis in acute-on-chronic liver failure},
author = {Ye Liu and Zihong Bao and Ju Zhou and Dingchun Li and Wu Li},
year = {2026},
journal = {Clinical and Experimental Hepatology},
doi = {10.5114/ceh.2026.164208},
url = {https://doi.org/10.5114/ceh.2026.164208}
}RIS
TY - JOUR TI - Th17/IL-17 may promote ferroptosis through modulation of the TP53/Nrf2 signaling axis in acute-on-chronic liver failure AU - Ye Liu AU - Zihong Bao AU - Ju Zhou AU - Dingchun Li AU - Wu Li PY - 2026 JO - Clinical and Experimental Hepatology DO - 10.5114/ceh.2026.164208 UR - https://doi.org/10.5114/ceh.2026.164208 ER -
APA
Liu, Y., Bao, Z., Zhou, J., Li, D., & Li, W. (2026). Th17/IL-17 may promote ferroptosis through modulation of the TP53/Nrf2 signaling axis in acute-on-chronic liver failure. Clinical and Experimental Hepatology. https://doi.org/10.5114/ceh.2026.164208
Source records
- crossref · retrieved 2026-11-10T23:53:03.665Z