Dataset: Prenatal iron deficiency (ID) exacerbates the risk of sudden infant death syndrome (SIDS) by modulating the expression of the hypothalamic orexin system, impairing homeostatic arousal mechanisms during hypoxia. - PathMap Experiment #000080
- DOI
- 10.5281/zenodo.21500678
- Published
- 2026
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- Not recorded
- Publisher
- Zenodo
- Open access
- yes
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BibTeX
@article{allodium:10.5281/zenodo.21500678,
title = {Dataset: Prenatal iron deficiency (ID) exacerbates the risk of sudden infant death syndrome (SIDS) by modulating the expression of the hypothalamic orexin system, impairing homeostatic arousal mechanisms during hypoxia. - PathMap Experiment \#000080},
author = {Dungan, Joshua},
year = {2026},
doi = {10.5281/zenodo.21500678},
url = {https://doi.org/10.5281/zenodo.21500678}
}RIS
TY - JOUR TI - Dataset: Prenatal iron deficiency (ID) exacerbates the risk of sudden infant death syndrome (SIDS) by modulating the expression of the hypothalamic orexin system, impairing homeostatic arousal mechanisms during hypoxia. - PathMap Experiment #000080 AU - Dungan, Joshua PY - 2026 DO - 10.5281/zenodo.21500678 UR - https://doi.org/10.5281/zenodo.21500678 ER -
APA
Joshua, D. (2026). Dataset: Prenatal iron deficiency (ID) exacerbates the risk of sudden infant death syndrome (SIDS) by modulating the expression of the hypothalamic orexin system, impairing homeostatic arousal mechanisms during hypoxia. - PathMap Experiment #000080. https://doi.org/10.5281/zenodo.21500678