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35 merged results for "Biased Signaling"

Partial results: at least one source did not answer. Available results are shown rather than treating an upstream outage as zero matches.

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  1. Structural Basis for Angiotensin II Type I Receptor G Protein-biased Signaling

    Author unknown · 2026 · bioRxiv

    uncertain Transparent signal score 50/100 · policy 1.0.0

    Found in europe-pmc · no DOI

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    • cautionMetadata completeness: 4 of 6 scored descriptive metadata groups are present; missing fields increase uncertainty. Source: Normalized work metadata; license: Caller-provided; provenance license not supplied to scorer
  2. Cancer cell‐intrinsic type 1 interferon: production, signaling, and outcomes within sex‐biased, female malignancies

    Ashlyn Conant, Nora Badiner, Sharon Asariah, Kiera McGivney · 2026 · Molecular Oncology

    uncertain Transparent signal score 53/100 · policy 1.0.0

    Found in doaj · DOI 10.1002/1878-0261.70311

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    • cautionMetadata completeness: 5 of 6 scored descriptive metadata groups are present; missing fields increase uncertainty. Source: Normalized work metadata; license: Caller-provided; provenance license not supplied to scorer
  3. Agonism and Biased Signaling.

    Kenakin T. · 2026 · Handb Exp Pharmacol

    limited evidence Transparent signal score 43/100 · policy 1.0.0

    Found in europe-pmc · DOI 10.1007/164_2025_770

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    • cautionMetadata completeness: 5 of 6 scored descriptive metadata groups are present; missing fields increase uncertainty. Source: Normalized work metadata; license: Caller-provided; provenance license not supplied to scorer
  4. GPCR Biased Signaling in Cancer.

    Avigad AC, Zhou M, Sun C, Ma L · 2026 · Handb Exp Pharmacol

    limited evidence Transparent signal score 43/100 · policy 1.0.0

    Found in europe-pmc · DOI 10.1007/164_2025_773

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    • cautionMetadata completeness: 5 of 6 scored descriptive metadata groups are present; missing fields increase uncertainty. Source: Normalized work metadata; license: Caller-provided; provenance license not supplied to scorer
  5. GPCR Biased Signaling in Metabolism.

    Zhang Z, Li Z. · 2026 · Handb Exp Pharmacol

    limited evidence Transparent signal score 43/100 · policy 1.0.0

    Found in europe-pmc · DOI 10.1007/164_2025_774

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    • cautionMetadata completeness: 5 of 6 scored descriptive metadata groups are present; missing fields increase uncertainty. Source: Normalized work metadata; license: Caller-provided; provenance license not supplied to scorer
  6. Novel pathways in gonadotropin receptor signaling and biased agonism

    Alfredo Ulloa-Aguirre, Pascale Crépieux, Anne Poupon, Marie-Christine Maurel · 2011 · Reviews in Endocrine and Metabolic Disorders

    limited evidence Transparent signal score 43/100 · policy 1.0.0

    Found in hal · DOI 10.1007/s11154-011-9176-2

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    • cautionMetadata completeness: 5 of 6 scored descriptive metadata groups are present; missing fields increase uncertainty. Source: Normalized work metadata; license: Caller-provided; provenance license not supplied to scorer
  7. Pharmacological profile of engineered 5-HT4 receptors and identification of 5-HT4 receptor-biased ligands

    Florence Gaven, Lucie P. Pellissier, Emilie Queffeulou, Maud Cochet-Bissuel · 2013 · Brain Research

    limited evidence Transparent signal score 45/100 · policy 1.0.0

    Found in hal · DOI 10.1016/j.brainres.2012.11.005

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    • cautionMetadata completeness: 5 of 6 scored descriptive metadata groups are present; missing fields increase uncertainty. Source: Normalized work metadata; license: Caller-provided; provenance license not supplied to scorer
  8. Biased Signaling of the Mu Opioid Receptor Revealed in Native Neurons

    Aliza T. Ehrlich, Meriem Semache, Florence Gross, Dillon da Fonte · 2019 · iScience

    limited evidence Transparent signal score 45/100 · policy 1.0.0

    Found in hal · DOI 10.1016/j.isci.2019.03.011

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    • cautionMetadata completeness: 5 of 6 scored descriptive metadata groups are present; missing fields increase uncertainty. Source: Normalized work metadata; license: Caller-provided; provenance license not supplied to scorer
  9. Biased GPCR signaling by the native parathyroid hormone–related protein 1 to 141 relative to its N-terminal fragment 1 to 36

    Karina A Peña, Alex D White, Sofya Savransky, Ignacio Portales Castillo · 2022 · Journal of Biological Chemistry

    limited evidence Transparent signal score 45/100 · policy 1.0.0

    Found in hal · DOI 10.1016/j.jbc.2022.102332

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    • cautionMetadata completeness: 5 of 6 scored descriptive metadata groups are present; missing fields increase uncertainty. Source: Normalized work metadata; license: Caller-provided; provenance license not supplied to scorer
  10. GPCR Biased Signaling: Opportunities and Challenges.

    Roth BL. · 2026 · Biochemistry

    limited evidence Transparent signal score 43/100 · policy 1.0.0

    Found in europe-pmc · DOI 10.1021/acs.biochem.6c00433

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    • cautionMetadata completeness: 5 of 6 scored descriptive metadata groups are present; missing fields increase uncertainty. Source: Normalized work metadata; license: Caller-provided; provenance license not supplied to scorer
  11. Molecular insights into intrinsic transducer-coupling bias in the CXCR4-CXCR7 system

    Parishmita Sarma, Carlo Marion C. Carino, Deeksha Seetharama, Shubhi Pandey · 2023 · Nature Communications

    uncertain Transparent signal score 53/100 · policy 1.0.0

    Found in doaj · DOI 10.1038/s41467-023-40482-9

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    • cautionMetadata completeness: 5 of 6 scored descriptive metadata groups are present; missing fields increase uncertainty. Source: Normalized work metadata; license: Caller-provided; provenance license not supplied to scorer
  12. Biased signaling of endothelial GPCRs as a therapeutic frontier in atherosclerosis.

    Chowdhury D, Mukherjee S, Dubey P, Paliwal SK · 2026 · J Recept Signal Transduct Res

    limited evidence Transparent signal score 43/100 · policy 1.0.0

    Found in europe-pmc · DOI 10.1080/10799893.2026.2748609

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    • not scoredOpen access status: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
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    • cautionMetadata completeness: 5 of 6 scored descriptive metadata groups are present; missing fields increase uncertainty. Source: Normalized work metadata; license: Caller-provided; provenance license not supplied to scorer
  13. Biased signaling regulates the pleiotropic effects of the urotensin II receptor to modulate its cellular behaviors

    Cédric Brulé, Nicolas Perzo, Jane-Eileen Joubert, Xavier Sainsily · 2014 · FASEB Journal

    limited evidence Transparent signal score 43/100 · policy 1.0.0

    Found in hal · DOI 10.1096/fj.14-249771

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    • cautionDOI registered: No matching Crossref record was present in this response. Source: Crossref; license: CC0 metadata
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    • not scoredDirectory of Open Access Journals: No matching DOAJ record was present in this response. No allow-list match; this is not evidence of low credibility. Source: DOAJ; license: CC0
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    • not scoredRetraction Watch retraction: No retraction notice matched this DOI in the deployed snapshot. No matching event found; coverage may be incomplete. Source: Retraction Watch; license: CC BY 4.0
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    • not scoredOpen access status: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredPublication license: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredPublication version: A publication version was supplied but is not scored. Source: Normalized work metadata; license: Caller-provided; provenance license not supplied to scorer
    • cautionMetadata completeness: 5 of 6 scored descriptive metadata groups are present; missing fields increase uncertainty. Source: Normalized work metadata; license: Caller-provided; provenance license not supplied to scorer
  14. The Role of Vitamin D and Vitamin D Receptor in Immunity to Leishmania major Infection

    James P. Whitcomb, Mary DeAgostino, Mark Ballentine, Jun Fu · 2012 · Journal of Parasitology Research

    uncertain Transparent signal score 53/100 · policy 1.0.0

    Found in doaj · DOI 10.1155/2012/134645

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    • not scoredRetraction Watch retraction: No retraction notice matched this DOI in the deployed snapshot. No matching event found; coverage may be incomplete. Source: Retraction Watch; license: CC BY 4.0
    • not scoredRetraction Watch expression of concern: No expression of concern notice matched this DOI in the deployed snapshot. No matching event found; coverage may be incomplete. Source: Retraction Watch; license: CC BY 4.0
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    • supportingOpen access status: Normalized open-access status: open. Source: Normalized work metadata; license: Caller-provided; provenance license not supplied to scorer
    • not scoredPublication license: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredPublication version: A publication version was supplied but is not scored. Source: Normalized work metadata; license: Caller-provided; provenance license not supplied to scorer
    • cautionMetadata completeness: 4 of 6 scored descriptive metadata groups are present; missing fields increase uncertainty. Source: Normalized work metadata; license: Caller-provided; provenance license not supplied to scorer
  15. Biased signaling of β2-adrenergic receptor in physiological and pathological states.

    Xu Y, Kawuribi V, Adu-Amankwaah J, Gong Z · 2026 · Cell Mol Biol Lett

    limited evidence Transparent signal score 45/100 · policy 1.0.0

    Found in europe-pmc · DOI 10.1186/s11658-026-00976-y

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    • supportingOpen access status: Normalized open-access status: open. Source: Normalized work metadata; license: Caller-provided; provenance license not supplied to scorer
    • not scoredPublication license: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredPublication version: A publication version was supplied but is not scored. Source: Normalized work metadata; license: Caller-provided; provenance license not supplied to scorer
    • cautionMetadata completeness: 5 of 6 scored descriptive metadata groups are present; missing fields increase uncertainty. Source: Normalized work metadata; license: Caller-provided; provenance license not supplied to scorer
  16. Integrated methylome and transcriptome analysis provides insight into DNA methylation-mediated networks in sexual dimorphism of Vernicia montana

    Xiang Dong, Lin Zhang, Huaguo Zhu, Jinping Peng · 2026 · BMC Plant Biology

    uncertain Transparent signal score 53/100 · policy 1.0.0

    Found in doaj · DOI 10.1186/s12870-026-09376-y

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    • not scoredRetraction Watch retraction: No retraction notice matched this DOI in the deployed snapshot. No matching event found; coverage may be incomplete. Source: Retraction Watch; license: CC BY 4.0
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    • supportingOpen access status: Normalized open-access status: open. Source: Normalized work metadata; license: Caller-provided; provenance license not supplied to scorer
    • not scoredPublication license: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredPublication version: A publication version was supplied but is not scored. Source: Normalized work metadata; license: Caller-provided; provenance license not supplied to scorer
    • cautionMetadata completeness: 5 of 6 scored descriptive metadata groups are present; missing fields increase uncertainty. Source: Normalized work metadata; license: Caller-provided; provenance license not supplied to scorer
  17. Targeting of specific CCR5-G protein complexes underlies biased signaling by HIV-1 envelope glycoproteins

    Romain Gasser, Philippe Colin, Célien Jacquemard, Théo Montagné · 2025 · Cell Communication and Signaling

    uncertain Transparent signal score 53/100 · policy 1.0.0

    Found in doaj · DOI 10.1186/s12964-025-02508-1

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    • not scoredMEDLINE indexed: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
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    • not scoredKnown publisher allow-list: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredROR affiliation: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredRetraction Watch retraction: No retraction notice matched this DOI in the deployed snapshot. No matching event found; coverage may be incomplete. Source: Retraction Watch; license: CC BY 4.0
    • not scoredRetraction Watch expression of concern: No expression of concern notice matched this DOI in the deployed snapshot. No matching event found; coverage may be incomplete. Source: Retraction Watch; license: CC BY 4.0
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    • supportingOpen access status: Normalized open-access status: open. Source: Normalized work metadata; license: Caller-provided; provenance license not supplied to scorer
    • not scoredPublication license: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredPublication version: A publication version was supplied but is not scored. Source: Normalized work metadata; license: Caller-provided; provenance license not supplied to scorer
    • cautionMetadata completeness: 5 of 6 scored descriptive metadata groups are present; missing fields increase uncertainty. Source: Normalized work metadata; license: Caller-provided; provenance license not supplied to scorer
  18. Spexin-GALR2-Gq axis as a biased signaling pathway for colitis therapy.

    Xue JW, Zhang T, Kong XL, Zheng K · 2026 · Zool Res

    limited evidence Transparent signal score 45/100 · policy 1.0.0

    Found in europe-pmc · DOI 10.24272/j.issn.2095-8137.2025.489

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    • not scoredROR affiliation: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredRetraction Watch retraction: No retraction notice matched this DOI in the deployed snapshot. No matching event found; coverage may be incomplete. Source: Retraction Watch; license: CC BY 4.0
    • not scoredRetraction Watch expression of concern: No expression of concern notice matched this DOI in the deployed snapshot. No matching event found; coverage may be incomplete. Source: Retraction Watch; license: CC BY 4.0
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    • supportingOpen access status: Normalized open-access status: open. Source: Normalized work metadata; license: Caller-provided; provenance license not supplied to scorer
    • not scoredPublication license: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredPublication version: A publication version was supplied but is not scored. Source: Normalized work metadata; license: Caller-provided; provenance license not supplied to scorer
    • cautionMetadata completeness: 5 of 6 scored descriptive metadata groups are present; missing fields increase uncertainty. Source: Normalized work metadata; license: Caller-provided; provenance license not supplied to scorer
  19. Contribution of IL-1RI Signaling to Protection against Cryptococcus neoformans 52D in a Mouse Model of Infection

    Mitra Shourian, Mitra Shourian, Ben Ralph, Ben Ralph · 2018 · Frontiers in Immunology

    uncertain Transparent signal score 53/100 · policy 1.0.0

    Found in doaj · DOI 10.3389/fimmu.2017.01987

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    • not scoredROR affiliation: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredRetraction Watch retraction: No retraction notice matched this DOI in the deployed snapshot. No matching event found; coverage may be incomplete. Source: Retraction Watch; license: CC BY 4.0
    • not scoredRetraction Watch expression of concern: No expression of concern notice matched this DOI in the deployed snapshot. No matching event found; coverage may be incomplete. Source: Retraction Watch; license: CC BY 4.0
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    • supportingOpen access status: Normalized open-access status: open. Source: Normalized work metadata; license: Caller-provided; provenance license not supplied to scorer
    • not scoredPublication license: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredPublication version: A publication version was supplied but is not scored. Source: Normalized work metadata; license: Caller-provided; provenance license not supplied to scorer
    • cautionMetadata completeness: 5 of 6 scored descriptive metadata groups are present; missing fields increase uncertainty. Source: Normalized work metadata; license: Caller-provided; provenance license not supplied to scorer
  20. Characterization of full-length long noncoding RNAs and identification of virus-responsive lncRNAs in Sogatella furcifera

    Yawen Ban, Yawen Ban, Ting Cui, Ting Cui · 2025 · Frontiers in Microbiology

    uncertain Transparent signal score 53/100 · policy 1.0.0

    Found in doaj · DOI 10.3389/fmicb.2025.1643735

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    • not scoredROR affiliation: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredRetraction Watch retraction: No retraction notice matched this DOI in the deployed snapshot. No matching event found; coverage may be incomplete. Source: Retraction Watch; license: CC BY 4.0
    • not scoredRetraction Watch expression of concern: No expression of concern notice matched this DOI in the deployed snapshot. No matching event found; coverage may be incomplete. Source: Retraction Watch; license: CC BY 4.0
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    • not scoredRetraction Watch reinstatement: No reinstatement notice matched this DOI in the deployed snapshot. No matching event found; coverage may be incomplete. Source: Retraction Watch; license: CC BY 4.0
    • supportingOpen access status: Normalized open-access status: open. Source: Normalized work metadata; license: Caller-provided; provenance license not supplied to scorer
    • not scoredPublication license: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredPublication version: A publication version was supplied but is not scored. Source: Normalized work metadata; license: Caller-provided; provenance license not supplied to scorer
    • cautionMetadata completeness: 5 of 6 scored descriptive metadata groups are present; missing fields increase uncertainty. Source: Normalized work metadata; license: Caller-provided; provenance license not supplied to scorer
  21. Stretch-Induced Biased Signaling in Angiotensin II Type 1 and Apelin Receptors for the Mediation of Cardiac Contractility and Hypertrophy

    Kinya Seo, Victoria N. Parikh, Euan A. Ashley, Euan A. Ashley · 2020 · Frontiers in Physiology

    uncertain Transparent signal score 53/100 · policy 1.0.0

    Found in doaj · DOI 10.3389/fphys.2020.00181

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    • not scoredROR affiliation: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredRetraction Watch retraction: No retraction notice matched this DOI in the deployed snapshot. No matching event found; coverage may be incomplete. Source: Retraction Watch; license: CC BY 4.0
    • not scoredRetraction Watch expression of concern: No expression of concern notice matched this DOI in the deployed snapshot. No matching event found; coverage may be incomplete. Source: Retraction Watch; license: CC BY 4.0
    • not scoredRetraction Watch correction: No correction notice matched this DOI in the deployed snapshot. No matching event found; coverage may be incomplete. Source: Retraction Watch; license: CC BY 4.0
    • not scoredRetraction Watch reinstatement: No reinstatement notice matched this DOI in the deployed snapshot. No matching event found; coverage may be incomplete. Source: Retraction Watch; license: CC BY 4.0
    • supportingOpen access status: Normalized open-access status: open. Source: Normalized work metadata; license: Caller-provided; provenance license not supplied to scorer
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    • not scoredPublication version: A publication version was supplied but is not scored. Source: Normalized work metadata; license: Caller-provided; provenance license not supplied to scorer
    • cautionMetadata completeness: 5 of 6 scored descriptive metadata groups are present; missing fields increase uncertainty. Source: Normalized work metadata; license: Caller-provided; provenance license not supplied to scorer
  22. Targeting the Angiotensin II Type 1 Receptor in Cerebrovascular Diseases: Biased Signaling Raises New Hopes

    Céline Delaitre, Michel Boisbrun, Sandra Lecat, François Dupuis · 2021 · International Journal of Molecular Sciences

    limited evidence Transparent signal score 45/100 · policy 1.0.0

    Found in hal · DOI 10.3390/ijms22136738

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    • supportingOpen access status: Normalized open-access status: open. Source: Normalized work metadata; license: Caller-provided; provenance license not supplied to scorer
    • not scoredPublication license: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredPublication version: A publication version was supplied but is not scored. Source: Normalized work metadata; license: Caller-provided; provenance license not supplied to scorer
    • cautionMetadata completeness: 5 of 6 scored descriptive metadata groups are present; missing fields increase uncertainty. Source: Normalized work metadata; license: Caller-provided; provenance license not supplied to scorer
  23. 45/60 NANOROBOT-ASSISTED AUTONOMOUS ORGAN-ON-CHIP MANUFACTURING AT THE LIMIT NANOROBOT-ASSISTED AUTONOMOUS ORGAN-ON-CHIP MANUFACTURING AT THE LIMIT is a 777-page English-language Research Design Working Edition in the STARLI Micro/Nanorobot Fabrication and Regenerative Biomanufacturing Sixty-Flagship series (Flagship 45/60). It investigates function requalification after independent device reconstruction. The research coupling is organ-on-chip fabrication x robotic placement x cross-laboratory functional reproduction. All proposed work is restricted to nonclinical in-vitro, organ-on-chip, synthetic phantom or authorized ex-vivo environments. No experiments were executed in preparing this edition; no human treatment, clinical efficacy or regulatory clearance is claimed. Its falsifiable candidate is: A locked chip manufacturing protocol will improve complete cross-site functional qualification versus commercial and manually seeded designs under reagent swaps. The Atlas minimum demonstrator is: Have two laboratories reproduce a liver-gut chip and exchange masked test samples across sites. Strong comparators include commercial organ-on-chip products and expert manual cell seeding. A central rival account is shared critical reagents or centralized calibration mistaken for reproducible autonomy. The nonnegotiable stop condition is: a hidden central supplier is required or barrier function cannot be rebuilt. Each failed, aborted, hidden-fault and incomplete run is retained in the experimental denominator, and observation of a finished form is never substituted for independently measured function. Thirty mathematical plates address domain-specific metrics and established model families across transport, measurement, elasticity, time-dependent dynamics, causal attribution, calibration and uncertainty. Eighty-two chapters and 410 separately paginated scientific investigations organize six research arcs. Two hundred Research Gates have been revised with independent observables, one of fifteen blinded causal intervention families, orthogonal controls and noncompensatory refusal criteria. Thirty mathematical plates now carry model-specific physical derivation commentary, explicit applicability limits and distinct flagship-level blind checks. Thirty-three Phoenix Answer Embryos propose unbuilt future instruments and succession artifacts, each requiring its own bench validation. This version is a research design and not a report of validated physical findings. It draws scientific method and conceptual boundaries from the user-supplied Atlas and research mother volumes; especially the Atlas-supplied ancestry DOI 10.5281/zenodo.23046134 and 10.5281/zenodo.23486020. Full external bibliographic DOI checking and scientific peer review have not been completed. Domain-specific sensitivity, causal identifiability and independent replication require qualified laboratory evaluation before any scientific superiority claim. Author: Feng Cheng-en (33) x STARLI / Xingli Series: STARLI Nano Regenerative Biomanufacturing / Flagship 45 of 60 Edition: Research Design Working Edition v1.1 / 2 November 2026 Join the STARLI Research Community Explore more than 100 interdisciplinary research themes and follow the evolving STARLI Research Gate community: https://zenodo.org/communities/starli-research-gate/

    33 · 2026

    limited evidence Transparent signal score 47/100 · policy 1.0.0

    Found in datacite, datacite · DOI 10.5281/zenodo.23864347

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    • not scoredROR affiliation: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredRetraction Watch retraction: No retraction notice matched this DOI in the deployed snapshot. No matching event found; coverage may be incomplete. Source: Retraction Watch; license: CC BY 4.0
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    • not scoredRetraction Watch reinstatement: No reinstatement notice matched this DOI in the deployed snapshot. No matching event found; coverage may be incomplete. Source: Retraction Watch; license: CC BY 4.0
    • supportingOpen access status: Normalized open-access status: open. Source: Normalized work metadata; license: Caller-provided; provenance license not supplied to scorer
    • supportingPublication license: A publication license was supplied: https://creativecommons.org/licenses/by/4.0/legalcode. Presence improves reuse transparency, not research validity. Source: Normalized work metadata; license: Caller-provided; provenance license not supplied to scorer
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  24. STARLI | F091–F100 / 300 Flagship 13 Fission · Volume I · Collective Microrobotic Autonomy, Swarm Safety and Mission Continuity Author: 33 | Nonclinical Research Design Working Edition v1.3 | English Zenodo Ten-Volume Overview This ten-volume collection closes the first hundred of the STARLI 300-theme research-design atlas. Research Unit I (F091–F095) examines autonomous sampling, local-to-global coordination, pheromone-like signaling, collision avoidance and collective energy allocation. Research Unit II (F096–F100) examines distributed planning, localization uncertainty, changing network topology, fault isolation and safe mission handoff. Each volume states prospective physical tests rather than completed experimental findings. The ten English working editions total 7,770 PDF pages: 300 candidate mathematical plates, 820 chapter openings, 4,100 independently paginated investigations, 2,000 Research Gates and 330 Phoenix Answer Embryos. The v1.3 Gate-annotation pass contrasts mechanisms, physical witnesses, blinded falsifiers, rivals, stopping boundaries and successor interfaces. These editorial counts and text-screening results do not establish causal independence or scientific validation. The Ten Research Directions F091 Nanorobotic Collective Autonomy Sampling At The Limit — Proposes controlled tests of swarm sampling throughput and identity preservation. A concealed communication partition probes whether completion gains survive the loss of collective coordination, against a centralized full-state controller; collision events and consensus error remain explicit vetoes. F092 Microrobotic Local Collaboration And Global Mission At The Limit — Asks whether agent-to-agent collaboration improves global agreement when robots lack an unmetered global-state feed. Hidden adversarial-neighbor states test dependence on outside tracking; fixed behavior rules with matched energy provide the comparator. F093 Nanorobotic Collective Pheromone Communication At The Limit — Explores prospective chemical-trail or signal-mediated coordination with collision exposure as a primary outcome. Hidden actuator saturation and a matched-actuation single-agent baseline challenge claims that signaling, rather than redundancy or imposed motion, caused the improvement. F094 Microscale Robot Collective Collision Avoidance Control At The Limit — Examines collision-safe motion while monitoring spatial coverage, recovery time and communication burden. A blinded shared-localization bias tests whether apparent avoidance depends on erroneous common position estimates; safety-boundary crossings trigger rejection. F095 Nanorobotic Collective Energy Allocation At The Limit — Tests dynamic allocation of limited energy across agents during dropout and recovery. Hidden agent-loss clusters challenge energy accounting and recovery-time claims against fixed rules with an equal energy budget; persistent unsafe confidence is a stop condition. F096 Microrobotic Distributed Mission Planning At The Limit — Studies task planning without a central controller using communication load, work-per-task and selective abstention as prospective metrics. Hidden network partitions expose plans that depend on unavailable messages, while a matched-actuation single-agent system tests whether coordination adds value. F097 Nanorobotic Collective Localization Uncertainty At The Limit — Tests whether robots can act safely when position confidence is uncertain or neighbors provide deceptive state information. Energy per accepted task, abstention and completion are checked against a full-state centralized benchmark; unrecognized safety-boundary crossings invalidate the claim. F098 Microrobotic Collective Dynamic Topology Control At The Limit — Examines when changing neighbor connections should trigger reconfiguration or justified refusal to proceed. Hidden actuator saturation challenges topology decisions; fixed energy-matched rules and independent task and consensus measurements distinguish adaptive control from misleading internal agreement. F099 Nanorobotic Collective Fault Isolation At The Limit — Investigates recognition and containment of malfunctioning agents without allowing errors to propagate across the swarm. A blinded shared-localization bias tests whether fault labels confuse a common sensor defect with separate robot failures; recovery to a known-safe state is mandatory. F100 Microrobotic Collective Mission Handoff At The Limit — Designs a traceable transfer of task state and authority across agents after concealed dropout clusters. The study checks consensus error, collisions and coverage against centralized full-state control; an apparently successful handoff is rejected if safety invariants are violated. Scope, Falsifiability and Evidence Boundaries All ten volumes are prospective, nonclinical research-design working editions for traceable materials, authorized ex-vivo systems, synthetic phantoms and bench-scale microfluidic tests. Proposed comparisons require preregistered budgets, blinded perturbations, independent reference measurements, complete negative-trial denominators and explicit safety stops. No new experimental results, validated in-body autonomy or clinical benefit are claimed. Mathematical derivations, physical unit consistency, claim-level DOI linkage and substantive causal novelty still require specialist review; the automatic Gate-text distinctness audit cannot replace it. Selected Established Methodological References Yang & Zhang (2021) · Motion Control in Magnetic Microrobotics: From Individual and Multiple Robots to Swarms · DOI 10.1146/annurev-control-032720-104318 | Farivarnejad & Berman (2022) · Multirobot Control Strategies for Collective Transport · DOI 10.1146/annurev-control-042920-095844 | STARLI Flagship 13 mother-method ancestry · DOI 10.5281/zenodo.23318840 These identifiers and methodological descriptions come from the supplied source-archive evidence register. They are reference points for existing methods, not evidence that any STARLI-specific candidate device or proposed experimental advantage has been demonstrated. Independent source-to-claim verification remains necessary. Join the STARLI Research Community Explore more than 100 interdisciplinary research themes and follow the evolving STARLI Research Gate community: https://zenodo.org/communities/starli-research-gate/ We do not need to know which door future generations will choose to open. We only hope that when they open it, they will find a little light to guide their way.

    33 · 2026

    limited evidence Transparent signal score 47/100 · policy 1.0.0

    Found in datacite, datacite · DOI 10.5281/zenodo.23915848

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  25. STARLI / TEN FLAGSHIP RESEARCH CONSTELLATION YULI · NANO × QUANTUM × REGENERATION × AI × BCI F001–F005 / 200 | Neural Nanomaterials | 777-page research working editions F001 Intelligent Nanomaterials for Neural Interfaces at the Limit Research identity: Charge-transfer integrity in compliant nanointerfaces. Endpoint: interfacial impedance [ohm]. Concealed discriminator: saline reference exchange. Conventional rival: standard platinum microelectrode matched for exposed area and recording bandwidth. F002 Programmable Neural Nanocomposites at the Limit Research identity: Programmability versus material heterogeneity. Endpoint: effective composite modulus [Pa]. Concealed discriminator: masked alignment reversal. Conventional rival: composition-matched nonprogrammable nanocomposite and classical Voigt/Reuss bounds. F003 Adaptive Conductive Nanonetworks for Neural Systems at the Limit Research identity: Adaptive conduction without hidden compensation. Endpoint: network conductance [S]. Concealed discriminator: pulse-duration scrambling. Conventional rival: fixed-resistance mesh and conventional redundant electrode network. F004 Ultraflexible Nanoelectrodes for BCI at the Limit Research identity: Flexibility must preserve electrical identity. Endpoint: fracture strain [1]. Concealed discriminator: unfamiliar soft phantom stiffness. Conventional rival: commercial flexible polymer electrode matched for electrical area and bending history. F005 Neural Tissue-Compatible Nanosurface Engineering at the Limit Research identity: Biological compatibility is not surface appearance. Endpoint: viable-cell fraction [%]. Concealed discriminator: wettability-matched control. Conventional rival: polished inert control and surface-chemistry-matched conventional coating. THE THREE-LETTER RESEARCH GATE PROTOCOL Each book proposes 30 distinct model functions, 82 chapters / 410 investigations, 200 independent test-entry contracts and 33 Phoenix study artifacts. Its Gates are letters to future researchers: Origin, Extreme Extrapolation and Future Research Plan. Literary possibility never substitutes for direct measurement. Evidence boundary: the YULI two-hundred-theme Word fixes each topic. STARLI F02 (DOI: 10.5281/zenodo.23486020) provides analogical engineering methods, not neurological efficacy evidence. External peer-reviewed materials and neural-interface methods remain separate sources.   STARLI / TEN FLAGSHIP RESEARCH CONSTELLATION YULI · NANO × QUANTUM × REGENERATION × AI × BCI F006–F010 / 200 | Neural Nanomaterials | 777-page research working editions F006 Low-Inflammation Neural Nanointerfaces at the Limit Research identity: Low inflammation requires direct cellular evidence. Endpoint: inflammatory cytokine change [pg/mL]. Concealed discriminator: low-dose thermal staircase. Conventional rival: inert surface and commercially established low-reactivity reference under equal culture conditions. F007 Self-Healing Neural Nanoconductors at the Limit Research identity: Repaired conductance is not restored safety. Endpoint: conductance recovery [%]. Concealed discriminator: thermal stress after restoration. Conventional rival: qualified redundant conductor and conventional replaceable electrode trace. F008 Signal-Selective Neural Nanomaterials at the Limit Research identity: Signal selectivity must survive irrelevant sources. Endpoint: target-to-offtarget amplitude ratio [dB]. Concealed discriminator: independent signal generator. Conventional rival: matched passive electrode with conventional analogue/digital bandpass filtering. F009 Long-Term Stable Neural Nanoimplant Materials at the Limit Research identity: Long-term stability must be witnessed over time. Endpoint: time-to-impedance-failure [day]. Concealed discriminator: late-cycle blind inspection. Conventional rival: conventional chronic-interface material with same ageing dose and endpoint thresholds. F010 Biodegradable Neural Interface Nanomaterials at the Limit Research identity: Biodegradability cannot conceal a lost function. Endpoint: dry-mass retention [%]. Concealed discriminator: second-lot monomer source. Conventional rival: stable reference interface and biodegradable polymer of established composition. SOURCE AND QUALITY CHARTER The 10 books are prospective nonclinical design working editions. Their original conjectures require independent experiments; unknown DOI identities, quantitative causal independence and professional mathematical review must not be reported as passed. Each volume preserves strong conventional baselines, blinded perturbations and hard-stop conditions. External reference examples: Tang et al., Nature Electronics (2023), DOI 10.1038/s41928-022-00913-9; Liu et al., Microsystems & Nanoengineering (2023), DOI 10.1038/s41378-022-00479-8; Shen et al., Nature Biomedical Engineering (2023), DOI 10.1038/s41551-023-01021-5. Join the STARLI Research Community · https://zenodo.org/communities/starli-research-gate/ We do not need to know which door future generations will choose to open. We only hope that when they open it, they will find a little light to guide their way. 🌈🚪✨

    33 · 2026

    limited evidence Transparent signal score 47/100 · policy 1.0.0

    Found in datacite, datacite · DOI 10.5281/zenodo.23965398

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  26. STARLI / YULI — F145/200 — Family 15: Regenerative BCI Co-Design 再生腦機介面微環境調控極限 Microenvironment Control In Regenerative BCI At The Limit Research position. This prospective, nonclinical Research Working Edition investigates Microenvironment oxygen concentration deviation [mg/L] in a hydrogel-electrode coculture testbed with a calibrated, independently acquired measurement reference. It proposes a scaffold–electrode–tissue causal model; it does not report completed experiments, animal studies, clinical efficacy, or validated regeneration. Testable claim. With acquisition time, authorized specimens, electrode exposure and reference assays matched, a preregistered joint-interface candidate is compared against an independently tuned conventional scaffold with a separate nonregenerative electrode. The masked challenge is concealed restriction of microfluidic flow; the chief rival is oxygen probe positioning causes apparent gradients. The primary candidate relation is Delta_C_O2 = C_target - C_measured. Interpretation requires Target and measured concentrations must share sampling time, spatial point, temperature and dissolved-oxygen calibration. Research architecture. The volume contains 30 mathematical and engineering diagnostic plates, 82 chapters with 410 one-page investigations, 200 Research Gates with three scientific letters per gate, and 33 Phoenix Answer Embryos. Methods include impedance and material-interface metrology, orthogonal biological witnesses, preregistered blinded tests, uncertainty propagation, negative-result retention, and independent reconstruction. Limitations and evidence. Escalating inflammation, premature material disintegration, loss of independent electrical reference, or revoked sample authorization are hard stops. Candidate mechanisms and formulas are not new confirmed physical laws. Foundational source identity was checked for Yuk, H., Wu, J., Zhao, X. (Nature Reviews Materials 7, 935-952 (2022)), DOI 10.1038/s41578-022-00483-4; Bierman-Duquette, R. D. et al. (Advanced Healthcare Materials 11(7), e2101577 (2022)), DOI 10.1002/adhm.202101577; these publications do not validate the new integrated hypothesis. Deep semantic distinctness across chapters and Gates requires specialist review. Author: 33 / STARLI–YULI Research Institute 36. Version v1.0; 2026-11-06. Join the STARLI Research Community Explore more than 100 interdisciplinary research themes and follow the evolving STARLI Research Gate community: https://zenodo.org/communities/starli-research-gate/

    33 · 2026

    limited evidence Transparent signal score 47/100 · policy 1.0.0

    Found in datacite, datacite · DOI 10.5281/zenodo.24017718

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  27. STARLI / TEN-FLAGSHIP RESEARCH CONSTELLATION YULI · NANO × QUANTUM × REGENERATION × AI × BCI F011–F015 / 200 | Family 02: Neural Regeneration Scaffolds F011 Nanofibrous Neural Regeneration Scaffolds at the Limit Core: nanofiber anisotropy × neurite extension × reference-controlled cytocompatibility. Endpoint: neurite extension length [micrometer]. Blind challenge: masked fiber-axis rotation. Strong baseline: randomly oriented polymer nanofiber and flat inert substrate under identical culture conditions. F012 Programmable Axonal Guidance Nanostructures at the Limit Core: reversible cue programming × axonal directionality × orthogonal geometry readout. Endpoint: axonal directional coherence [dimensionless]. Blind challenge: hidden groove-angle crossover. Strong baseline: static patterned guidance grooves and unpatterned stiffness-matched gel. F013 Neural Stem Cell Nanomicroenvironments at the Limit Core: extracellular matrix cues × stem-cell identity × longitudinal differentiation tracing. Endpoint: lineage-marker-positive cell fraction [percent]. Blind challenge: concealed integrin-blocking dose. Strong baseline: defined-medium monolayer and composition-matched nonstructured hydrogel. F014 Nanotopology and Synaptic Reconstruction at the Limit Core: surface nanotopology × synaptic apposition × electrophysiology confirmation. Endpoint: synaptic colocalization density [per square millimeter]. Blind challenge: concealed nanoridge wavelength. Strong baseline: flat and microscale-patterned controls with independent synaptic-functional recordings. F015 Conductive Hydrogels for Neural Regeneration at the Limit Core: hydrated network conduction × tissue-compatible mechanics × independent neural readout. Endpoint: scaffold bulk conductivity [siemens per meter]. Blind challenge: hidden filler-percolation fraction. Strong baseline: composition-matched nonconductive hydrogel and commercial electrode under equal hydration. SCIENTIFIC INHERITANCE AND GATE CONTRACT Each volume follows the 777-page research book architecture: 14 distinct front-matter arguments, 30 formulas, 82 chapters and 410 individual investigations, 200 experimentally discriminating Research Gates and 33 Phoenix candidates. The three-letter prose is a gift to future scientists, not evidence that an experiment occurred. The source F02 publication supplies engineering rules for distinguishing apparent recovery from verified function. It is not evidence of neuronal regeneration. The YULI atlas fixes the ten independent research themes.   STARLI / TEN-FLAGSHIP RESEARCH CONSTELLATION YULI · NANO × QUANTUM × REGENERATION × AI × BCI F016–F020 / 200 | Family 02: Neural Regeneration Scaffolds F016 Intelligent Remodeling of Neural Injury Microenvironments at the Limit Core: bounded milieu feedback × inflammation markers × independent cellular phenotype. Endpoint: cytokine reduction in culture [nanogram per milliliter]. Blind challenge: masked cytokine spiking. Strong baseline: static intervention and vehicle-matched hydrogel with the same cell count. F017 Self-Assembling 3D Neural Nanoscaffolds at the Limit Core: programmable assembly forces × 3D pore topology × qualified cell ingress. Endpoint: connected pore volume fraction [percent]. Blind challenge: masked fiber-length distribution. Strong baseline: templated 3D porous scaffold and static porogen fabrication with matched solids. F018 Bioelectric-Coupled Neural Nanoscaffolds at the Limit Core: electric field exposure × axon steering × electrochemical dose restraint. Endpoint: field-linked neurite direction change [degree]. Blind challenge: concealed polarity reversal. Strong baseline: unstimulated matched scaffold and conventional low-field electrode control. F019 Scaffold Degradation and Neural Function Preservation at the Limit Core: enzymatic scaffold erosion × mechanical support transfer × cellular function qualification. Endpoint: function retained during degradation [percent]. Blind challenge: masked enzyme activity titration. Strong baseline: nondegrading scaffold and controlled-rate degradable reference matched for initial stiffness. F020 Cross-Laboratory Validation of Neural Regeneration Scaffolds at the Limit Core: batch and site effects × metrological traceability × blind external reconstruction. Endpoint: between-laboratory standardized effect contrast [dimensionless standardized difference]. Blind challenge: blind site-label permutation. Strong baseline: standardized two-site ring trial and single-site frozen-protocol baseline. EVIDENCE AND SCIENTIFIC LIMITS These are prospective, nonclinical working editions. Efficacy, model causality and long-term safety require independent experiments. Published methods (Bierman-Duquette et al., DOI 10.1002/adhm.202101577; Yuk et al., DOI 10.1038/s41578-022-00483-4; Dawit et al., DOI 10.1039/D4BM00048J) do not validate these new design hypotheses. Join the STARLI Research Community · https://zenodo.org/communities/starli-research-gate/ We do not need to know which door future generations will choose to open. We only hope that when they open it, they will find a little light to guide their way. 🌈🚪✨

    33 · 2026

    limited evidence Transparent signal score 47/100 · policy 1.0.0

    Found in datacite, datacite · DOI 10.5281/zenodo.24047053

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  28. The Wicked Problem of Selection and Competency-Based Education: Applying TRIZ Inversion to the Graduate Medical Education Selection Process

    Holly A. Caretta-Weyer, Ming-Ka Chan, Mary Ellen J. Goldhamer, Alan Schwartz · 2026 · Perspectives on Medical Education

    uncertain Transparent signal score 53/100 · policy 1.0.0

    Found in doaj · DOI 10.5334/pme.2596

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  29. Biased signaling via NtsR1 restrains food intake and weight gain in obese mice

    Ramírez-Virella J, Black K, Dennis NF, Bugescu R · 2026

    limited evidence Transparent signal score 39/100 · policy 1.0.0

    Found in europe-pmc · DOI 10.64898/2026.08.30.748142

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    • cautionMetadata completeness: 5 of 6 scored descriptive metadata groups are present; missing fields increase uncertainty. Source: Normalized work metadata; license: Caller-provided; provenance license not supplied to scorer
  30. Structural Basis for Angiotensin II Type I Receptor G Protein-biased Signaling

    Liu J, Skiba MA, Kruse AC. · 2026

    limited evidence Transparent signal score 39/100 · policy 1.0.0

    Found in europe-pmc · DOI 10.64898/2026.10.13.758900

    Show all credibility signals
    • cautionDOI registered: No matching Crossref record was present in this response. Source: Crossref; license: CC0 metadata
    • cautionDOI resolves: No matching Crossref record was present in this response. Source: Crossref; license: CC0 metadata
    • not scoredDirectory of Open Access Journals: No matching DOAJ record was present in this response. No allow-list match; this is not evidence of low credibility. Source: DOAJ; license: CC0
    • not scoredMEDLINE indexed: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredOpenAlex core source: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredKnown publisher allow-list: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredROR affiliation: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredRetraction Watch retraction: No retraction notice matched this DOI in the deployed snapshot. No matching event found; coverage may be incomplete. Source: Retraction Watch; license: CC BY 4.0
    • not scoredRetraction Watch expression of concern: No expression of concern notice matched this DOI in the deployed snapshot. No matching event found; coverage may be incomplete. Source: Retraction Watch; license: CC BY 4.0
    • not scoredRetraction Watch correction: No correction notice matched this DOI in the deployed snapshot. No matching event found; coverage may be incomplete. Source: Retraction Watch; license: CC BY 4.0
    • not scoredRetraction Watch reinstatement: No reinstatement notice matched this DOI in the deployed snapshot. No matching event found; coverage may be incomplete. Source: Retraction Watch; license: CC BY 4.0
    • not scoredOpen access status: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredPublication license: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • cautionPublication version: Identified as a preprint; peer review and later versions may change the record. Source: Normalized work metadata; license: Caller-provided; provenance license not supplied to scorer
    • cautionMetadata completeness: 5 of 6 scored descriptive metadata groups are present; missing fields increase uncertainty. Source: Normalized work metadata; license: Caller-provided; provenance license not supplied to scorer
  31. Correct setup of the substantia nigra requires Reelin-mediated fast, laterally-directed migration of dopaminergic neurons

    Ankita Ravi Vaswani, Beatrice Weykopf, Cathleen Hagemann, Hans-Ulrich Fried · 2019 · eLife

    uncertain Transparent signal score 53/100 · policy 1.0.0

    Found in doaj · DOI 10.7554/elife.41623

    Show all credibility signals
    • cautionDOI registered: No matching Crossref record was present in this response. Source: Crossref; license: CC0 metadata
    • cautionDOI resolves: No matching Crossref record was present in this response. Source: Crossref; license: CC0 metadata
    • supportingDirectory of Open Access Journals: A matching record was returned by DOAJ. Source: DOAJ; license: CC0
    • not scoredMEDLINE indexed: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredOpenAlex core source: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredKnown publisher allow-list: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredROR affiliation: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredRetraction Watch retraction: No retraction notice matched this DOI in the deployed snapshot. No matching event found; coverage may be incomplete. Source: Retraction Watch; license: CC BY 4.0
    • not scoredRetraction Watch expression of concern: No expression of concern notice matched this DOI in the deployed snapshot. No matching event found; coverage may be incomplete. Source: Retraction Watch; license: CC BY 4.0
    • not scoredRetraction Watch correction: No correction notice matched this DOI in the deployed snapshot. No matching event found; coverage may be incomplete. Source: Retraction Watch; license: CC BY 4.0
    • not scoredRetraction Watch reinstatement: No reinstatement notice matched this DOI in the deployed snapshot. No matching event found; coverage may be incomplete. Source: Retraction Watch; license: CC BY 4.0
    • supportingOpen access status: Normalized open-access status: open. Source: Normalized work metadata; license: Caller-provided; provenance license not supplied to scorer
    • not scoredPublication license: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredPublication version: A publication version was supplied but is not scored. Source: Normalized work metadata; license: Caller-provided; provenance license not supplied to scorer
    • cautionMetadata completeness: 5 of 6 scored descriptive metadata groups are present; missing fields increase uncertainty. Source: Normalized work metadata; license: Caller-provided; provenance license not supplied to scorer
  32. Biased Signaling at the 5-HT7 Receptor: Novel Therapeutic Agents for Pain Modulation

    Séverine Morisset-Lopez · 2025

    limited evidence Transparent signal score 48/100 · policy 1.0.0

    Found in hal · no DOI

    Show all credibility signals
    • not scoredDOI registered: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredDOI resolves: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredDirectory of Open Access Journals: No matching DOAJ record was present in this response. No allow-list match; this is not evidence of low credibility. Source: DOAJ; license: CC0
    • not scoredMEDLINE indexed: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredOpenAlex core source: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredKnown publisher allow-list: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredROR affiliation: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredRetraction Watch retraction: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredRetraction Watch expression of concern: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredRetraction Watch correction: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredRetraction Watch reinstatement: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredOpen access status: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredPublication license: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredPublication version: A publication version was supplied but is not scored. Source: Normalized work metadata; license: Caller-provided; provenance license not supplied to scorer
    • cautionMetadata completeness: 5 of 6 scored descriptive metadata groups are present; missing fields increase uncertainty. Source: Normalized work metadata; license: Caller-provided; provenance license not supplied to scorer
  33. KINETIC BIASED AND COMPARTMENTALIZED SIGNALING : A SYSTEM BIOLOGY APPROACH

    Romain Yvinec · 2024

    uncertain Transparent signal score 50/100 · policy 1.0.0

    Found in hal · no DOI

    Show all credibility signals
    • not scoredDOI registered: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredDOI resolves: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredDirectory of Open Access Journals: No matching DOAJ record was present in this response. No allow-list match; this is not evidence of low credibility. Source: DOAJ; license: CC0
    • not scoredMEDLINE indexed: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredOpenAlex core source: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredKnown publisher allow-list: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredROR affiliation: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredRetraction Watch retraction: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredRetraction Watch expression of concern: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredRetraction Watch correction: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredRetraction Watch reinstatement: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • supportingOpen access status: Normalized open-access status: open. Source: Normalized work metadata; license: Caller-provided; provenance license not supplied to scorer
    • not scoredPublication license: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredPublication version: A publication version was supplied but is not scored. Source: Normalized work metadata; license: Caller-provided; provenance license not supplied to scorer
    • cautionMetadata completeness: 5 of 6 scored descriptive metadata groups are present; missing fields increase uncertainty. Source: Normalized work metadata; license: Caller-provided; provenance license not supplied to scorer
  34. KINETIC BIASED AND COMPARTMENTALIZED SIGNALING : A SYSTEM BIOLOGY APPROACH

    Romain Yvinec · 2025

    uncertain Transparent signal score 50/100 · policy 1.0.0

    Found in hal · no DOI

    Show all credibility signals
    • not scoredDOI registered: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredDOI resolves: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredDirectory of Open Access Journals: No matching DOAJ record was present in this response. No allow-list match; this is not evidence of low credibility. Source: DOAJ; license: CC0
    • not scoredMEDLINE indexed: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredOpenAlex core source: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredKnown publisher allow-list: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredROR affiliation: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredRetraction Watch retraction: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredRetraction Watch expression of concern: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredRetraction Watch correction: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredRetraction Watch reinstatement: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • supportingOpen access status: Normalized open-access status: open. Source: Normalized work metadata; license: Caller-provided; provenance license not supplied to scorer
    • not scoredPublication license: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredPublication version: A publication version was supplied but is not scored. Source: Normalized work metadata; license: Caller-provided; provenance license not supplied to scorer
    • cautionMetadata completeness: 5 of 6 scored descriptive metadata groups are present; missing fields increase uncertainty. Source: Normalized work metadata; license: Caller-provided; provenance license not supplied to scorer
  35. Probing heterotrimeric G protein activation: applications to biased ligands.

    Colette Denis, Aude Saulière, Ségolène Galandrin, Jean-Michel Sénard · 2012 · Current Pharmaceutical Design

    uncertain Transparent signal score 50/100 · policy 1.0.0

    Found in hal · no DOI

    Show all credibility signals
    • not scoredDOI registered: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredDOI resolves: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredDirectory of Open Access Journals: No matching DOAJ record was present in this response. No allow-list match; this is not evidence of low credibility. Source: DOAJ; license: CC0
    • not scoredMEDLINE indexed: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredOpenAlex core source: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredKnown publisher allow-list: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredROR affiliation: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredRetraction Watch retraction: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredRetraction Watch expression of concern: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredRetraction Watch correction: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredRetraction Watch reinstatement: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • supportingOpen access status: Normalized open-access status: open. Source: Normalized work metadata; license: Caller-provided; provenance license not supplied to scorer
    • not scoredPublication license: Not checked or no result supplied; no credibility inference made. Source: No authority result supplied; license: Unknown
    • not scoredPublication version: A publication version was supplied but is not scored. Source: Normalized work metadata; license: Caller-provided; provenance license not supplied to scorer
    • cautionMetadata completeness: 5 of 6 scored descriptive metadata groups are present; missing fields increase uncertainty. Source: Normalized work metadata; license: Caller-provided; provenance license not supplied to scorer